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Providers / Bioss Primary Conjugated Antibodies. ALEXA FLUOR / Bcl-2 (Ser70) Antibody, ALEXA FLUOR 594

Description:

Size: 100ul

Catalog no.: bs-3052R-A594

Price: 350 EUR

Get for 350 EUR on Gentaur.com

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Product details

Gene ID Number

24224

Modification Site

Ser70

Swiss Prot

P49950

Subcellular location

Cytoplasm

Immunogen range

43-93/236

French translation

anticorps

Crossreactivity

Mouse, Rat

Clonality

Polyclonal

Target Antigen

Bcl-2 Ser70

Excitation emission

590nm/617nm

Concentration

1ug per 1ul

Modification

Phosphorylation

Conjugated with

ALEXA FLUOR® 594

Conjugated

Alexa conjugate 1

Tested applications

IF(IHC-P), IF(ICC)

Clone

Polyclonal antibody

Purification

Purified by Protein A.

Conjugation

Alexa Fluor,ALEXA FLUOR® 594

Category

Conjugated Primary Antibodies

Host Organism

Rabbit (Oryctolagus cuniculus)

Also known as

Anti-Bcl-2 Ser70 PAb ALEXA FLUOR 594

Recommended dilutions

IF(IHC-P)(1:50-200), IF(ICC)(1:50-200)

Synonyms

Bcl-2; Apoptosis regulator Bcl-2; Bcl2

Specificity

This is a highly specific antibody against Bcl-2 Ser70.

Long name

Bcl-2 (Ser70) Polyclonal Antibody, ALEXA FLUOR 594 Conjugated

Cross-reactive species details

Due to limited amount of testing and knowledge, not every possible cross-reactivity is known.

Source

KLH conjugated synthetic phosphopeptide derived from rat Bcl-2 around the phosphorylation site of Ser70

Storage conditions

Store this antibody in aqueous buffered solution containing 1% BSA, 50% glycerol and 0.09% sodium azide. Keep refrigerated at 2 to 8 degrees Celcius for up to one year.

Properties

For facs or microscopy Alexa 1 conjugate.If you buy Antibodies supplied by Bioss Primary Conjugated Antibodies. ALEXA FLUOR they should be stored frozen at - 24°C for long term storage and for short term at + 5°C.

Background of the antigen

Suppresses apoptosis in a variety of cell systems including factor-dependent lymphohematopoietic and neural cells. Regulates cell death by controlling the mitochondrial membrane permeability. Appears to function in a feedback loop system with caspases. Inhibits caspase activity either by preventing the release of cytochrome c from the mitochondria and/or by binding to the apoptosis-activating factor (APAF-1).