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Providers / Bioss Primary Conjugated Antibodies. ALEXA FLUOR / APOC3 Antibody, ALEXA FLUOR 594

Description:

Size: 100ul

Catalog no.: bs-5037R-A594

Price: 380 EUR

Get for 380 EUR on Gentaur.com

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Product details

Modification Site

None

Gene ID Number

11814

Target Antigen

APOC3

Swiss Prot

P33622

Subcellular location

Secreted

Tested applications

IF(IHC-P)

French translation

anticorps

Crossreactivity

Mouse, Rat

Modification

Unmodified

Clonality

Polyclonal

Immunogen range

55-100/101

Concentration

1ug per 1ul

Excitation emission

590nm/617nm

Conjugated with

ALEXA FLUOR® 594

Conjugated

Alexa conjugate 1

Recommended dilutions

IF(IHC-P)(1:50-200)

Clone

Polyclonal antibody

Purification

Purified by Protein A.

Conjugation

Alexa Fluor,ALEXA FLUOR® 594

Category

Conjugated Primary Antibodies

Also known as

Anti-APOC3 PAb ALEXA FLUOR 594

Host Organism

Rabbit (Oryctolagus cuniculus)

Specificity

This is a highly specific antibody against APOC3.

Long name

APOC3 Polyclonal Antibody, ALEXA FLUOR 594 Conjugated

Synonyms

apo-CIII; apoC-III; Apolipoprotein C-III; Apolipoprotein C3; Apoc3

Source

KLH conjugated synthetic peptide derived from rat Apolipoprotein CIII

Cross-reactive species details

Due to limited amount of testing and knowledge, not every possible cross-reactivity is known.

Storage conditions

Store this antibody in aqueous buffered solution containing 1% BSA, 50% glycerol and 0.09% sodium azide. Keep refrigerated at 2 to 8 degrees Celcius for up to one year.

Properties

For facs or microscopy Alexa 1 conjugate.If you buy Antibodies supplied by Bioss Primary Conjugated Antibodies. ALEXA FLUOR they should be stored frozen at - 24°C for long term storage and for short term at + 5°C.

Background of the antigen

Component of triglyceride-rich very low density lipoproteins (VLDL) and high density lipoproteins (HDL) in plasma. Plays a multifaceted role in triglyceride homeostasis. Intracellularly, promotes hepatic very low density lipoprotein 1 (VLDL1) assembly and secretion; extracellularly, attenuates hydrolysis and clearance of triglyceride-rich lipoproteins (TRLs). Impairs the lipolysis of TRLs by inhibiting lipoprotein lipase and the hepatic uptake of TRLs by remnant receptors.