Description:

Size: 100ul

Catalog no.: bs-6915R-A350

Price: 380 EUR

Product details

Modification Site

None

Gene ID Number

26053

Target Antigen

AUTS2

Tested applications

IF(IHC-P)

French translation

anticorps

Clonality

Polyclonal

Modification

Unmodified

Concentration

1ug per 1ul

Excitation emission

343nm/442nm

Conjugated with

ALEXA FLUOR® 350

Crossreactivity

Human, Mouse, Rat

Clone

Polyclonal antibody

Recommended dilutions

IF(IHC-P)(1:50-200)

Purification

Purified by Protein A.

Conjugation

Alexa Fluor,ALEXA FLUOR 350

Category

Conjugated Primary Antibodies

Host Organism

Rabbit (Oryctolagus cuniculus)

Also known as

Anti-AUTS2 PAb ALEXA FLUOR 350

Specificity

This is a highly specific antibody against AUTS2.

Long name

AUTS2 Polyclonal Antibody, ALEXA FLUOR 350 Conjugated

Cross-reactive species details

Due to limited amount of testing and knowledge, not every possible cross-reactivity is known.

Synonyms

Autism related protein 1; Autism susceptibility candidate 2; Autism susceptibility gene 2 protein; FBRSL2.

Source

This antibody was obtained by immunization of the host with KLH conjugated synthetic peptide derived from human AUTS2

Storage conditions

Store this antibody in aqueous buffered solution containing 1% BSA, 50% glycerol and 0.09% sodium azide. Keep refrigerated at 2 to 8 degrees Celcius for up to one year.

Properties

For facs or microscopy Alexa 1 conjugate.Alexa Fluor 350 conjugates can be used in multi-color flow cytometry with FACS's equipped with a second red laser or red diode.If you buy Antibodies supplied by Bioss Primary Conjugated Antibodies. ALEXA FLUOR they should be stored frozen at - 24°C for long term storage and for short term at + 5°C.

Background of the antigen

Autism susceptibility gene 2 protein (AUTS2) is strongly expressed in brain, skeletal muscle and kidney and is also expressed in placenta, lung and leukocytes. There are 2 isoforms produced by alternative splicing; isoform long and isoform short of 139 kDa and 136 kDa respectively. This gene is interrupted by a translocation breakpoint in a pair of autistic twins.