Description:

Size: 100ul

Catalog no.: bs-2001R-A594

Price: 380 EUR

Product details

Modification Site

None

Crossreactivity

Virus

Immunogen range

20-70/566

Tested applications

IF(IHC-P)

French translation

anticorps

Modification

Unmodified

Clonality

Polyclonal

Excitation emission

590nm/617nm

Concentration

1ug per 1ul

Conjugated with

ALEXA FLUOR® 594

Conjugated

Alexa conjugate 1

Recommended dilutions

IF(IHC-P)(1:50-200)

Clone

Polyclonal antibody

Target Antigen

H1N1 Hemagglutinin 1

Purification

Purified by Protein A.

Cross-reactive species details

Influenza A virus H1N1

Category

Conjugated Primary Antibodies

Subcellular location

Membrane bound, Cell membrane

Conjugation

Alexa Fluor,ALEXA FLUOR® 594

Host Organism

Rabbit (Oryctolagus cuniculus)

Also known as

Anti-H1N1 Hemagglutinin 1 PAb ALEXA FLUOR 594

Specificity

This is a highly specific antibody against H1N1 Hemagglutinin 1.

Long name

H1N1 Hemagglutinin 1 Polyclonal Antibody, ALEXA FLUOR 594 Conjugated

Synonyms

HA; HA1; Hemagglutinin; Influenza A Virus [A/California/04/2009H1N1].

Source

KLH conjugated synthetic peptide derived from Influenza A Virus Hemagglutinin

Storage conditions

Store this antibody in aqueous buffered solution containing 1% BSA, 50% glycerol and 0.09% sodium azide. Keep refrigerated at 2 to 8 degrees Celcius for up to one year.

Properties

For facs or microscopy Alexa 1 conjugate.If you buy Antibodies supplied by Bioss Primary Conjugated Antibodies. ALEXA FLUOR they should be stored frozen at - 24°C for long term storage and for short term at + 5°C.

Background of the antigen

Influenza A virus is a major public health threat. Novel influenza virus strains caused by genetic drift and viral recombination emerge periodically to which humans have little or no immunity, resulting in devastating pandemics. Influenza A can exist in a variety of animals; however it is in birds that all subtypes can be found. These subtypes are classified based on the combination of the virus coat glycoproteins hemagglutinin (HA) and neuraminidase (NA) subtypes. During 1997, an H5N1 avian influenza virus was determined to be the cause of death in 6 of 18 infected patients in Hong Kong. There was some evidence of human to human spread of this virus, but it is thought that the transmission efficiency was fairly low. HA interacts with cell surface proteins containing oligosaccharides with terminal sialyl residues. Virus isolated from a human infected with the H5N1 strain in 1997 could bind to oligosaccharides from human as well as avian sources, indicating its species jumping ability.Influenza A Virus [A/California/04/2009(H1N1)]