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Providers / Bioss Primary Conjugated Antibodies. ALEXA FLUOR / Streptokinase Antibody, ALEXA FLUOR 350

Description:

Size: 100ul

Catalog no.: bs-9846R-A350

Price: 380 EUR

Get for 380 EUR on Gentaur.com

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Product details

Modification Site

None

Subcellular location

Secreted

Crossreactivity

Bacteria

Tested applications

IF(IHC-P)

French translation

anticorps

Clonality

Polyclonal

Modification

Unmodified

Concentration

1ug per 1ul

Excitation emission

343nm/442nm

Immunogen range

250-300/2876

Cross-reactive species details

Streptococcus

Target Antigen

Streptokinase

Conjugated with

ALEXA FLUOR® 350

Recommended dilutions

IF(IHC-P)(1:50-200)

Clone

Polyclonal antibody

Purification

Purified by Protein A.

Conjugation

Alexa Fluor,ALEXA FLUOR 350

Category

Conjugated Primary Antibodies

Host Organism

Rabbit (Oryctolagus cuniculus)

Also known as

Anti-Streptokinase PAb ALEXA FLUOR 350

Specificity

This is a highly specific antibody against Streptokinase.

Long name

Streptokinase Polyclonal Antibody, ALEXA FLUOR 350 Conjugated

Synonyms

ska; skc; skg; Kabikinase; Streptase; Kinalysin; Plasminokinase; SK.

Source

KLH conjugated synthetic peptide derived from Streptococcus Streptokinase

Storage conditions

Store this antibody in aqueous buffered solution containing 1% BSA, 50% glycerol and 0.09% sodium azide. Keep refrigerated at 2 to 8 degrees Celcius for up to one year.

Properties

For facs or microscopy Alexa 1 conjugate.Alexa Fluor 350 conjugates can be used in multi-color flow cytometry with FACS's equipped with a second red laser or red diode.If you buy Antibodies supplied by Bioss Primary Conjugated Antibodies. ALEXA FLUOR they should be stored frozen at - 24°C for long term storage and for short term at + 5°C.

Background of the antigen

Streptokinase is a metallo-enzyme produced by various strains of Streptococcus that can liquify blood clots by converting plasminogen to plasmin. Streptokinase is not a protease but it activates plasminogen by complexing with it. It is thought to prevent the formation of effective fibrin barriers around the site of infection, thereby contributing to the invasiveness of the cells.